17 beta hydroxysteroid dehydrogenase iii deficiency

The mechanism by which the glycyrrhizinates exert their effect on the renin-angiotensin-aldosterone system has been elucidated. 2 , 35 , 36 , 42 Competitive (and reversible) inhibition of the enzyme 11-beta-hydroxysteroid dehydrogenase results in the suppression of cortisol conversion to inactive cortisone. Consequent suppression of plasma renin activity and aldosterone levels is evident. Exchangeable sodium levels increase and cortisol occupation of mineralocorticoid receptors in the distal kidney tubules is enhanced. The condition responds to administration of spironolactone, potassium supplementation, and discontinuation of licorice.

17 beta hydroxysteroid dehydrogenase iii deficiency

17 beta hydroxysteroid dehydrogenase iii deficiency

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17 beta hydroxysteroid dehydrogenase iii deficiency17 beta hydroxysteroid dehydrogenase iii deficiency17 beta hydroxysteroid dehydrogenase iii deficiency17 beta hydroxysteroid dehydrogenase iii deficiency17 beta hydroxysteroid dehydrogenase iii deficiency

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